demonstrated that silencing of HCs TAZ in murine models of nonalcoholic steatohepatitis (NASH) prevented or reversed hepatic inflammation, HCs death, and fibrosis 36. signaling. Conclusions: Our work has Sarafloxacin HCl identified a hepatocyte-specific lnc-HSER that regulates liver fibrosis, providing a proof that this molecule is a novel biomarker for damaged HCs and a potential target…